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    However, effective treatment is available to treat most of the cases with tuberculosis. The response to the treatment is generally very good and gratifying but at times it may be poor and frustrating, e.g., in patients with coexistent HIV infection, infection with resistant strains of Mycobacterium tuberculosis, poor drug compliance, and 585 606 Garg and Tandon development of serious toxicity necessitating stoppage of drugs. Introduction Isoniazid INH Metabolism and Possible Mechanism of Hepatotoxicity Rifampicin Pyrazinamide Combination of Isoniazid and Rifampicin and Hepatotoxicity Risk Factors for ATT-Induced Hepatotoxicity Clinical Course of ATT-Induced Hepatotoxicity Management of ATT-Induced Hepatotoxicity Treatment of Underlying Tuberculosis Recommendation for Reintroduction of ATT Conclusion References I.

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