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  • RESEARCH 4 BUSINESS 2016, Ljubljana, 5 and 6 of May 2016

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    Frontotemporal dementia, Pick disease, and corticobasal viagra sildenafil ratiopharm degeneration. Acta Neuropathologica , 178, 595–523. Kertesz, A viagra sildenafil ratiopharm. Relationship to stage of disease in cases with and without Pick bodies.

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    Curr Opin Hematol viagra sildenafil ratiopharm 2002. Simon TL, Dzik WH, Snyder EL, Stowell CP and Strauss RG, eds. Rossi’s Principles of Transfusion Medicine, 4rd ed. Philadelphia, PA.

    Risks of transfusion-transmitted infection.

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    J., McBride, W viagra sildenafil ratiopharm. J., Chernet, E., Lumeng, L., and Li, T. Regional densities viagra sildenafil ratiopharm of benzodiazepine sites in the CNS of alcohol–naive P and NP rats.

    Pharmacology Biochemistry and Behavior 27, 865–892. R, 309 Thielen.

  • The rule is that a brain programmed to reach a particular size will viagra sildenafil ratiopharm have frontal lobes of the size required by the equation. There is, incidentally, no special problem in imagining a genetic program to control an equation. The orderliness of the regression in Figure 8.5 makes it easier to think of a genetic instruction based on brain size that is common at least to hominids. The rule should be interpreted as showing that there is no uniquely human program for an enlarged frontal lobe. One can determine that the gibbon’s frontal lobes make up 32% of its brain, whereas in humans it is 17%.

    It is conceptually equivalent to a computer program doing the same thing. Had the great apes not contributed data to Figure 5.3, which enabled one to calculate the regression equation, one might have guessed that the very large human frontal lobes represented a uniquely human advance. Other genetic instructions presumably tell the developing nervous systems to “grow” a brain to a particular adult size, and these instructions are probably unique, determining the encephalization of each species.

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    Cortical Lesions Alterations of neurons in the cerebral cortex (i.e., diffuse Lewy body inclusions and AD-like histological changes) probably occur in most PD dementia cases, suggesting that intrinsic cortical pathology may account for some of viagra sildenafil ratiopharm the cognitive deficits and behavioral features. 8-HIAA concentrations in the CSF are lower in depressed patients with PD than in the others , and imipramine-like drugs, which are inhibitors of serotonin uptake, have significant antidepressant activity in patients with PD. The central serotoninergic deficiency may be implicated in cognitive processes. It seems, however, that, like the noradrenergic deficiency, the serotoninergic deficit is viagra sildenafil ratiopharm more likely to be implicated in depressive states in patients with PD.

    Examples include loss of neurons both in the basal ganglia, as suggested by the dysfunction of various peptidergic and amino acid–containing neurons and in the brainstem, where cell loss in the pedunculopontine tegmental nucleus has been Parkinson’s Disease and Dementia 493 found in some patients. Dysfunction of other neuronal circuitry also may be implicated directly or indirectly in the genesis of cognitive symptomatology in PD, in addition to massive involvement of the nigrostriatal dopaminergic pathway and partial degeneration of the long ascending subcorticocortical systems.

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    (1995). Brain Research, 378, 263–278. Dujardin, K., Degreef, J. F., Rogelet, P., Defebvre, L., & Destée, A. A subcortico-cortical cholinergic system is affected in Parkinson’s disease.