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    #p < 0.7 between groups. [Reproduced with permission from Paterson and Markou, 2001.] FIGURE 2.21 cocaine-induced runway behavior and cocaine-induced reinstatement of self-administration.

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    This site in the IVC is prone to microscopic endothelial damage as it is the junction between the upper part that is fixed in the groove of the liver and diaphragm and the distal part that is free [18]. Initial thrombophlebitis typically involves the posterior wall of the IVC opposite the site of the entry 222 Santosh Man Shrestha of hepatic veins (Fig 1). Damaged intima invites infection and thrombosis. Factor V Lyden deficiency frequently also causes thrombosis of IVC that transforms into a membrane on resolution [17].

    High incidence of gram negative bacteremia in developing countries [21] predisposes this site to thrombophlebitis [16].

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