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  • RESEARCH 4 BUSINESS 2016, Ljubljana, 5 and 6 of May 2016

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  • An elegant study by Lewis and O’Donnell (1997) showed that in vivo stimulation of VTA with trains of pulses resembling VTA DA neuron burst firing evoked a sustained depolarization of PFC neurons that had very similar temporal and spatial features to an viagra mail spam up state. One hypothesis is that DA D1 receptor activation sustains and stabilizes up states by activating NMDA receptors, as well as yet unidentified ionic conductances (Wang & O’Donnell, 1997). 2000), seamans & Yang.

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    Primate studies have shown that D1-like receptors are primarily located on the dendritic spines and shafts of pyramidal neurons, and on both dendrites and axon terminals of GABA-ergic interneurons (Bergson, Mrzljak, Lidow, Goldman-Rakic, & Levenson, 1994), indicating that the effects of DA on PFC pyramidal neurons, via D1 receptor activation, are both direct, modulating the firing activity of PFC neurons, and also indirect, tuning the release of GABA onto them. Intra-VTA stimulation, leading primarily to DA release, might produce a variety of effects on PFC neurons, depending on whether PFC neurons are in the up or in the down state (for a comprehensive review, see O’Donnell, 2004.

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