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    4.34). The use of an asymmetric lesion technique to functionally dissect the role of the central nucleus of the amygdala and BNST revealed a critical, but not exclusive, role for the CRF pathway from the central nucleus of the amygdala to the BNST in footshock-induced reinstatement (Erb et al., 1998). Similarly, microinjection studies have shown a role for norepinephrine originating in the ventral noradrenergic pathway and projecting to the BNST, and the central nucleus of the amygdala also has a role in footshock-induced cocaine-related reinstatement (Leri et al., 1998. However, in the absence of cocaine administration, an environmental cue may induce craving and relapse through enhanced glutamate transmission with little dopamine involvement.

    [Reproduced with permission from Cornish and Kalivas, 1998.] opioid peptide role in stress-induced reinstatement of drug-related responding (Shalev et al., 1999). The brain site critical for the CRF role in footshockinduced reinstatement appears to be the bed nucleus of the stria terminalis (BNST), an area rich in CRF receptors, terminals, and cell bodies (Erb and Stewart, 2000) (Fig.

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