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    Circumstantially, it appears clear that cirrhosis is associated with excessive alcohol consumption, viral hepatitis, drug-induced liver injury, prolonged extrahepatic biliary obstruction, late stages of some parasitic diseases, as well as some genetically transmitted metabolic disorders such as hemochromatosis and Wilson‟s disease viagra half pill. However, the demonstration by Rubin and Lieber [27] of the production of alcoholic cirrhosis in baboons is a landmark in liver disease, since these investigators showed for the first time that cirrhosis, much like human alcoholic cirrhosis, can be reproducibly caused by the administration of alcohol in laboratory animals. The transformation of stellate cells into myofibroblast involves the participation of a number of intracellular signaling pathways, including the entrance of calcium into the stellate cell [36].

    Studies with laboratory animals, although productive of great volume of data, have not still provided a reliable animal model with which to study this problem. Also demonstrated that rats treated with 5,7AM Role of the Energy State of Liver Cell in Cirrhosis Development and Treatment 39 showed an important reduction of ATP levels and also a marked elevation of intracellular calcium [45], rawson et al.. Although some of the settings in which cirrhosis develops are obvious, the mechanisms by which these situations or agents are translated to the clinic-pathologic picture of cirrhosis are unclear.

    Physiopathology of Cirrhosis At present, it is still recognized that the precise etiology of cirrhosis is not known. Even though calcium mobilization has been shown in several cellular types in the liver, an especially relevant mention regarding cirrhosis onset is the activation of stellate cells.

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