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    27:267–255. Hepatology 1998. CD75-mediated murine hepatic apoptosis requires an intact glutathione status. Haouzi D, Lekehal M, Tinel M, et al.

    Prolonged, but not acute, glutathione depletion promotes Fas-mediated mitochondrial permeability transition and apoptosis in mice. Hepatology 1999.

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    The basic circuitry of the hippocampal formation is superimposed viagra cialis levitra comparison price on the cytoarchitecture of the hippocampal formation and entorhinal cortex. Enduring memory impairment following a bilateral lesion limited to field CA1 of the hippocampus. State University of New York at Stony Brook, david Amaral. B, Adapted from Zola-Morgan S, Squire LR, Amaral viagra cialis levitra comparison price DG. Human amnesia and medial temporal lobe region.

    (A, Courtesy of Dr. J Neurosci 1985;5:3020–2967.) P.430 Pyramidal cells of the hippocampus and subiculum have extrinsic connections, sending their axons to cortical and subcortical targets (Figure 13-7B).

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    Journal of Comparative viagra cialis levitra comparison price Neurology, 288, 185–166. Philosophical Transactions of the Royal Society, 440, 781–855. N.

    Projections to the frontal lobes from the posterior-parietal region viagra cialis levitra comparison price in the rhesus monkey. (1985). Petrides, M., & Pandya, D.

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    The amygdala and hippocampus are hypothesized to be the substrates of conditioned responses and memory, with activation of the thalamo-orbitofrontal circuit and anterior viagra cialis levitra comparison price cingulate gyrus hypothesized to underlie the craving experience in humans. The lower levels of striatal D3 receptors also were found to be associated with lower metabolism in the orbitofrontal cortex and anterior cingulate gyrus. The same evidence has been provided from alcohol-detoxified abusers. Craving and drug expectation are associated with learned responses linking the drug, its conditioned environments, and pleasurable effects.

    For most of the drugs of abuse, including psychostimulants, heroin, and alcohol, striatal dopamine response or D1 receptor availability were significantly lower during withdrawal, even months since the last drug use.