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    Whereas, IL-1β, venta viagra sildenafil capital federal 4, 6, TNF-α, and IFN-γ, appear to participate in the pathogenesis of the mild to severe inflammatory phenomena seen in alcoholic and post-hepatitis C liver cirrhosis, respectively. Throughout CDL the relationship among chronic hepatocellular damage, liver inflammation and cirrhosis has not been clearly defined, but cytokines could be the common link between these complex pathological outcomes (Llorente et al., 1992). Summarizing there is increasing evidence that several cytokines mediate the deleterious hepatic inflammation, apoptosis, and necrosis of liver cells, as well as the characteristic chronic processes cholestasis and fibrosis, previous to the end stage cirrhosis (Tilg, 1999). Also, data indicate that neither 12 Eduardo Fernández-Martínez IFN-γ nor IL-11, at least at the levels observed in post-hepatitis C liver cirrhosis, are able to counteract the fibrotic/inflammatory processes. Thalidomide Effects on Liver Damage and Cirrhosis Thalidomide Effects on Liver Damage Alcoholic liver disease is maybe one of the most important hepatopathies around the world.

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