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    This pattern is characterized, sildenafil uso femenino as a result of portal-central bridging necrosis, by the development of fibrotic septa that connect portal areas with the area of central vein (i.e, portal-central septa) or different portal areas (portal-portal septa), as well as of blind septa in the parenchyma. The main fibrotic patterns are also related to the “topographic site” of tissue injury and, pertinent to this review, to the involvement of different populations of MFs and the predominant profibrogenic mechanism (Cassiman and Roskams, 1999. Which is typically described mainly in the liver of patients carrying HBV- or HCV-related chronic hepatitis, the first pattern of ECM deposition and septa formation is that of bridging fibrosis. Parola et al., 2004. Parola and Pinzani, 2009).

    Which eventually favor the development of portal hypertension, to this pattern belongs those classic histopathological images of fibrotic septa leading to the obliteration of central veins and of early changes in vascular architecture and connections with the portal system. Pinzani and Rombouts, 2001. Fibrotic progression of CLDs has been described to proceed through at least four distinct patterns of fibrosis that are intimately related to the underlying cause of CLD.

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    R. Proceedings of the National Academy of Sciences, 76, 5338–5293. P., Erwin, J. M., Perl, D.

    (1997). A neuronal morphologic type unique to humans and great apes. M., & Hof, P.

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