• RESEARCH 4 BUSINESS 2016, Ljubljana, 5 and 6 of May 2016

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    One key pathological hallmark of the disease is granular osmiophilic material (GOM) in the smallvessel walls in the skin and brain, as well as in other areas (Peters, Opherk, sildenafil citrate canada Bergmann, et al., 2006). There is evidence from one postmortem case that there may be a cholinergic deficit in CADASIL. Patients’ initial cognitive symptoms involve executive function, problems with attention, processing speed and verbal fluency, and other frontal lobe tasks (Dichgans et al., 1999. Disease results from abnormalities in the wall of small vessels, although sildenafil citrate canada the precise mechanism is not fully elucidated.

    Peters, Opherk, Danek, et al., 2004). Even patients without dementia have subtle frontal lobe cognitive deficits that do not necessarily correlate with the amount of lesions on brain MRI. The white matter arteriopathy and resulting leukoencephalopathy and subcortical infarcts in CADASIL lead to mild cognitive impairment characterized by significant frontal–executive dysfunction, likely due to involvement of frontal–subcortical pathways eventually resulting over time in a vascular dementia syndrome.

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