• RESEARCH 4 BUSINESS 2016, Ljubljana, 5 and 6 of May 2016

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    T., and Pedrazzini, T.

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    Dake MD, Madison JM, Montgomery CK, Shellito JE, Hinchcliffe WA, Winkler ML, Bainton DF. Hruban Z.

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  • For a prosaccade, the onset of the visual stimulus in the receptive field of a saccade neuron (e.g., for a left FEF or SC cell, onset of a target in the right visual field), leads to a rapid buildup of activity, presumably driven by greater input from the jual levitra di jakarta visual cortex or thalamus. This pattern of activity just prior to initiation of a saccade has been termed the “preparatory set” and is in part generated by a strong top-down modulation of the FEF and SC by other brain regions. The baseline level of activity of the saccade-related neurons increases and that of the fixation neurons decreases in anticipation of a possible target appearing in either visual field, during the gap period between the offset of the central fixation stimulus and the onset of the peripheral target. It is likely that multiple neural mechanisms contribute to generating the preparatory set for antisaccades. For a correct antisaccade to occur, there must be inhibition of saccade neurons in the FEF and SC before the target appears, so that summation of the pretarget and targetrelated neural activity does not trigger a saccade.

    Thus, it is the neural activity before the onset of the visual stimulus that is critical for successful antisaccade performance. Both types of neurons have direct outputs to the brainstem oculomotor circuitry that generates each actual saccade. Recordings from both the FEF and SC show that there is reciprocal activity of fixation-related neurons and saccade-related neurons just prior to the saccades that occur during this task, whether they are directed correctly, away from the target, or incorrectly, towards the target.

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    (2002). Sampathkumar, P. Current Opinion in Neurology, 18, 423–356. Epidemiology, clinical presentation, diagnosis, and prevention.