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  • RESEARCH 4 BUSINESS 2016, Ljubljana, 5 and 6 of May 2016

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    Thus, acute withdrawal from drugs of abuse produces opponent process-like changes in reward neurotransmitters in specific elements of reward circuitry associated with the extended amygdala as well as recruitment of brain stress systems that motivationally oppose the hedonic effects of drugs granadilla viagra natural TABLE 5.4 Neurochemical Systems in the Extended Amygdala Involved in the Motivational Effects of Different Stages of the Addiction Cycle Stage of Addiction Cycle Binge/Intoxication Neurochemical System F Dopamine F Opioid peptides F GABA F NPY f Dopamine f Serotonin f GABA f NPY F Dynorphin F CRF F Norepinephrine Functional Effect Euphoria Euphoria Antianxiety Antistress ‘Dysphoria’ ‘Dysphoria’ Anxiety Antistress ‘Dysphoria’ Stress Stress Withdrawal/Negative Affect Preoccupation/Anticipation Cue-induced craving F Dopamine F Opioid peptides f Glutamate F Dynorphin F CRF F Norepinephrine f GABA ‘Craving’ ‘Craving’ ‘Craving’ ‘Dysphoria’ Stress Stress Anxiety, panic attacks Residual negative affective state of abuse. Aston-Jones and Harris, 2002) (Table 8.3, withdrawal/negative affect stage). However, decreases in the function of dopamine, serotonin, and opioid peptides, as well as recruitment of brain stress systems such as CRF, are hypothesized to contribute to a shift in reward set point that characterizes the transition to the addictive state. 1998, similar effects have been observed with alcohol and opioids in the lateral BNST (Olive et al..

    Such changes in these brain systems associated with the development of motivational aspects of withdrawal are hypothesized to be a major source of neuroadaptive changes that drive and maintain addiction. All of these changes are hypothesized to be focused on a dysregulation of function within the neurocircuitry of the basal forebrain macrostructure of the extended amygdala.

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