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  • But changes in certain intermediate early genes genuine viagra for sale with chronic drug exposure in animal models show the possibility of long-term changes in specific brain regions that may be common to all drugs of abuse, no biochemical markers are sufficiently specific to predict a given stage of the addiction cycle. Molecular studies have identified transduction and transcription factors that may mediate dependence-induced reward and stress dysregulation and chronic vulnerability changes in neurocircuitry associated with the development and maintenance of addiction. Both GABAergic and glutamatergic systems are directly or indirectly altered by acute administration of drugs of abuse in the ventral tegmental area or nucleus accumbens and provide a key substrate for the cellular neuroplasticity associated with chronic drug administration.

    Genetic studies in animals, to date, suggest roles for the genes encoding the neurochemical elements involved in brain reward (dopamine, opioid peptide), withdrawal (Mpdz), and stress (NPY) systems in the vulnerability to addiction. The brain stress systems in the extended amygdala are directly implicated in stress-induced relapse. Human imaging studies HOMEOSTASIS VERSUS ALLOSTASIS IN ADDICTION 465 reveal similar neurocircuits involved in acute intoxication, chronic drug dependence, and vulnerability to relapse.

    Two salient changes that cut across different drugs in established and unrecovered substance-dependent individuals are decreases in prefrontal cortex function and decreases in brain dopamine D2 receptors, while no exact imaging results necessarily predict addiction. Including activation of mesolimbic dopamine neurons and drug reward sensitive neurons in the nucleus accumbens and amygdala, electrophysiological studies have identified several common elements of acute drug reward.

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