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    NSAIDs developed in the past two decades often have had hepatic injury observed during clinical trials or soon after initial marketing because of the routine use of biochemical testing , in contrast. In addition, cellular unrest, ballooning, and eosinophilic degeneration have been described. Aspirin injury is both dose- and blood concentration–dependent consistent with intrinsic toxicity (27) as seen in both animals and cell culture experiments (28,31).

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