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    The effects of CREB in the nucleus accumbens have been hypothesized to involve increased expression of dynorphin, a neuropeptide associated with decreased function of the mesolimbic dopamine system, and dysphoria-like responses (Bals-Kubik et al., 1994). ‘There is now compelling evidence that upregulation of the cAMP pathway and CREB in the nucleus accumbens represents a mechanism of ‘’motivational tolerance and dependence’’…These molecular adaptations decrease an individual’s sensitivity to the rewarding effects of subsequent drug exposures (tolerance) and impair the reward pathway (dependence) so that after removal of the drug the individual is left in an amotivational, depressed-like state’ (Nestler, 2003).

  • 209:239–310. Int J Pharm 1997. 10:417–332. Regional differences and effect of weight reduction on human fat cell metabolism.

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