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    A moderate and asymptomatic phase, and a second phase in which neuronal loss becomes sufficient for memory deficits to appear. If these results can be transposed to humans, they suggest that disorders of cognition mediated by ascending subcorticocortical systems may be observed in PD, either when there is sufficient partial destruction of several ascending neuronal pathways, or when there is a severe and selective lesion of one of them. Age of onset of the disease may influence the threshold at which neuronal lesions become symptomatic.

    Intellectual impairment occurs when synaptic adjustments are no longer sufficient to compensate for neuronal loss.

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    Ann Emerg Med 1983;14:567–54.

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