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    There is a profound loss of glutamatergic neurons in frontal cortex in FTLD (Ernst, Chang, Melchor, & Mehringer, 1993), but no data suggest that the mechanism for this loss is via excessive excitotoxic activity at the level of N-methyl-D-aspartate (NMDA) cheap viagra sydney receptors. Trinh, Hoblyn, Mohanty, & Yaffe, 2004). Tariot et al., 1997. Therefore, we do cheap viagra sydney not recommend these compounds for either the cognitive or behavioral disorder associated with FTLD. However, there is no evidence for a cholinergic deficit associated with any subtypes FTLD, and in our experience, cholinesterase inhibitors can precipitate worsening of behavior.

    Despite the absence of a strong scientific rationale for NMDA-blockers, the recent successes with these compounds in slowing progression of AD (Reisberg et al., 2002.

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    And Weisbrodt, N.W. Castro, G.A., Behnke, J.M.

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