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    More than 65% of patients with halothane liver injury present within 3 weeks of exposure with fever, nausea, rash, arthralgias, and diffuse abdominal discomfort. Prognostic factors for poor outcome include a short latent period from exposure to jaundice, obesity, age older than 30 years, hepatic encephalopathy, and prolongation of the prothrombin time.

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    It is proposed that the motor subcircuit containing the projection from the anterior cingulate/prelimbic region of the dorsal prefrontal cortex (dPFC) to the core of the nucleus accumbens (NAcore) corresponds to a final common pathway for all priming stimuli. Parts of the medial prefrontal cortex and amygdala appear to be particularly involved in cue-induced reinstatement (Figs.

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