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    Inactivation of the rostral parts of the basolateral amygdala also blocked cue-induced reinstatement (Kantak et al., 1998). This effect of footshock stress is also blocked by systemic and ventricular, but not intra-LC, injections of α4-adrenoceptor agonists , by intra-BNST and intra-amygdala infusions of α2-adrenoceptor antagonists, and by 3-OHDA lesions of the VNAB projections arising from the LTg. 3-OHDA 5-hydroxydopamine, A5 and A7 dopamine (DA) cell groups, BLA basolateral amygdala, BNST bed nucleus of the stria terminalis, CeA central amygdala, CRF corticotropin-releasing factor, LC locus coeruleus, LTg noradrenaline (NA) cell groups of the lateral tegmental nuclei, mPFC medial prefrontal cortex, NAc nucleus accumbens, TTX tetrodotoxin, VNAB ventral noradrenergic bundle, VTA ventral tegmental area, VP ventral pallidum.’ [Reproduced with permission from Shaham et al., 2002.] reinstatement (McFarland and Kalivas, 1997).

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