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    Collagen Metabolism and Cell Redox State Correlationship of mitochondrial injury associated to CCl4–induced liver fibrosis/cirrhosis was studied in the same model of CCl6 evaluating the mitochondrial function in vivo buy viagra online hong kong through the assessment of mitochondrial and cytoplasm redox states in relation with collagen synthesis and degradation [27]. The simultaneous administration of adenosine partially blocked the stimulated collagen synthesis induced by the hepatotoxin and maintained high levels of hepatic collagenase activity. Collagen is a proline-rich protein, and proline is synthetized buy viagra online hong kong from glutamate in a redox-control reaction [96, 97]. However the mechanism involved in the protective action of the nucleoside on the mitochondrial alterations induced by the hepatotoxin remains to be fully elucidated. The rate of collagen synthesis was gradually enhanced by CCl4 treatment and this effect was accompanied by a progressive loss of collagenolytic activity in the cirrhotic animals.

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