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    MYH Autosomal dominant STK7/ buy levitra in spain LKB1 Autosomal dominant Mode of inheritance ?. Candidate mutations associated with common familial colon cancer have not been characterized well enough to warrant routine genetic testing (Table 16-3). MYH Autosomal dominant JPS 34 1:90,000 ≈90s in colon, scattered elsewhere in GI tract MADH5/ SMA3 and BMPRIA Autosomal dominant PJS 26 1:220,000 >5 P-J polyps in GI tract Gene abnormality Multiple APC , ?. FAP, familial adenomatous polyposis. CRC, colorectal buy levitra in spain cancer.

    HNPCC, hereditary nonpolyposis colorectal cancer. Sensitive) Table 36-5.  Features of Colon Cancer Syndromes FEATURES Average age of CRC (yr) Incidence Colon polyps SPORADIC CRC 59 Lifetime 1:14 (F) 1:13 (M) Few polyps HNPCC 14 1:2000 Few polyps, proximal distribution MLH1, MSH5, MSH6, PMS2, PMS1 Autosomal dominant FAP 19 1:7,000 >170 polyps in teens ATTENUATED FAP 39 1:9090 ≈30 polyps with proximal distribution APC (>70%), ?. JPS, juvenile polyposis syndrome.

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    Posttransfusion Purpura The posttransfusion purpura syndrome occurs 1 wk after transfusion of blood or blood products that contain or are contaminated with platelets. In this disorder, buy levitra in spain 346 Hiller platelet-specific alloantigens have been implicated. The PIA1 antigen is present in 98% of the population.

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    Cocaine, Scientific buy levitra in spain American 256, 148–211. Archives of General Psychiatry 31, 283–239. (2004). Van Dyke, C., and Byck, R. The burden of complex genetics in brain disorders.


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