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  • However, it buy cheapest cialis online is essential to find correlations with etiopathogenesis sources. Curri’s findings prove microcirculatory alterations accompanied by. Especially in nerve areas where stimulation favors lipolysis, merlen’s remarks on the increased volume of adipose cells and the diffusion distance between capillaries and fatty cells might explain the cause of trophic turbulences closely PATHOPHYSIOLOGY OF CELLULITE & 45 associated with vasculo–tissular exchange alterations. Such anatomic structure is essential to understand the etiopathogenesis of the liposclerosis diseases.

    This does not mean that cellulite starts as buy cheapest cialis online microcirculation pathology but that microcirculation is also affected in regions where the interstitial matrix basal regulation is altered. Alterations in arteriole flow account for Vage’s findings that ‘‘blood and lymphatic flow through adipose tissue are inversely proportional to its growth,’’ so that ‘‘slow circulation’’ involves ‘‘lipogenesis,’’ whereas ‘‘quick circulation’’ involves ‘‘lipolysis.’’ All these consequently result in therapeutic difficulties and lead to prolong ‘‘localized obesity.’’ Besides, this explains the lipolytic effects of carboxytherapy. & & & & & & Slowdown of microcirculation Venular stasis Microaneurysm Lipedema Alterations in venule capillary permeability GAG decrease in vascular sleeves The continuous alternation of ‘‘evening lipedema’’ through transpiration generated by capillary and postcapillary venule stasis leads to its final chronicity. Based on Curri’s findings, the relationship among capillaries (CAP), fat cell (CA), and matrix with fibroblast (F) was established.

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